Sargina Warda, I’m sure you meant normal as in no withdraw and not “high’. I don’t know anyone who gets high. threads like these are scanned looking for reasons to make kratom illegal in all states. a lot of people in chronic pain would be in a horrible position. so we should choose our words wisely. You can buy the gel capsules and a capsule filler on ebay. really cheap and fills 12 or more at a time. great for going out or every so often. 2 tsps would not be 20 caps but would still be quite a few. the acid in the OJ makes a difference and you would prob miss it. stick with licking a lawnmower LOL
In any case, there is nonetheless a good deal of evidence to indicate that modafinil is producing at least a portion of its wakefulness-promoting effects by acting as a DRI, or at least via activation of the dopaminergic system. In support of modafinil acting as a dopaminergic agent, its wakefulness-promoting effects are abolished in DAT knockout mice (although it is important to note that DAT knockout mice show D 1 and D 2 receptor and norepinephrine compensatory abnormalities, which might confound this finding), reduced by both D 1 and D 2 receptor antagonists (although conflicting reports exist),  and completely blocked by simultaneous inactivation of both D 1 and D 2 receptors.  In accordance, modafinil shows full stimulus generalization to other DAT inhibitors including cocaine, methylphenidate, and vanoxerine, and discrimination is blocked by administration of both ecopipam (SCH-39166), a D 1 receptor antagonist, and haloperidol , a D 2 receptor antagonist.  Partial substitution was seen with the DRA dextroamphetamine and the D 2 receptor agonist PNU-91356A , as well as with nicotine (which indirectly elevates dopamine levels through activation of nicotinic acetylcholine receptors ).